5-HT2A receptor and Prolactin

Shortcuts: Differences, Similarities, Jaccard Similarity Coefficient, References.

Difference between 5-HT2A receptor and Prolactin

5-HT2A receptor vs. Prolactin

The mammalian 5-HT2A receptor is a subtype of the 5-HT2 receptor that belongs to the serotonin receptor family and is a G protein-coupled receptor (GPCR). Prolactin (PRL), also known as luteotropic hormone or luteotropin, is a protein that is best known for its role in enabling mammals, usually females, to produce milk.

Similarities between 5-HT2A receptor and Prolactin

5-HT2A receptor and Prolactin have 7 things in common (in Unionpedia): Agonist, Antipsychotic, Central nervous system, Dopamine receptor D2, Oligodendrocyte, Oxytocin, Risperidone.

Agonist

An agonist is a chemical that binds to a receptor and activates the receptor to produce a biological response.

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Antipsychotic

Antipsychotics, also known as neuroleptics or major tranquilizers, are a class of medication primarily used to manage psychosis (including delusions, hallucinations, paranoia or disordered thought), principally in schizophrenia and bipolar disorder.

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Central nervous system

The central nervous system (CNS) is the part of the nervous system consisting of the brain and spinal cord.

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Dopamine receptor D2

Dopamine receptor D2, also known as D2R, is a protein that, in humans, is encoded by the DRD2 gene.

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Oligodendrocyte

Oligodendrocytes, or oligodendroglia,.

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Oxytocin

Oxytocin (Oxt) is a peptide hormone and neuropeptide.

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Risperidone

Risperidone, sold under the trade name Risperdal among others, is an antipsychotic medication.

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The list above answers the following questions

What 5-HT2A receptor and Prolactin have in common
What are the similarities between 5-HT2A receptor and Prolactin

5-HT2A receptor and Prolactin Comparison

5-HT2A receptor has 175 relations, while Prolactin has 175. As they have in common 7, the Jaccard index is 2.00% = 7 / (175 + 175).

References

This article shows the relationship between 5-HT2A receptor and Prolactin. To access each article from which the information was extracted, please visit:

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