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Autocrine signalling and Benign prostatic hyperplasia

Shortcuts: Differences, Similarities, Jaccard Similarity Coefficient, References.

Difference between Autocrine signalling and Benign prostatic hyperplasia

Autocrine signalling vs. Benign prostatic hyperplasia

Autocrine signaling is a form of cell signaling in which a cell secretes a hormone or chemical messenger (called the autocrine agent) that binds to autocrine receptors on that same cell, leading to changes in the cell. Benign prostatic hyperplasia (BPH), also called prostate enlargement, is a noncancerous increase in size of the prostate.

Similarities between Autocrine signalling and Benign prostatic hyperplasia

Autocrine signalling and Benign prostatic hyperplasia have 3 things in common (in Unionpedia): Cancer, Paracrine signalling, Transcription (biology).

Cancer

Cancer is a group of diseases involving abnormal cell growth with the potential to invade or spread to other parts of the body.

Autocrine signalling and Cancer · Benign prostatic hyperplasia and Cancer · See more »

Paracrine signalling

Paracrine signaling is a form of cell-to-cell communication in which a cell produces a signal to induce changes in nearby cells, altering the behavior of those cells.

Autocrine signalling and Paracrine signalling · Benign prostatic hyperplasia and Paracrine signalling · See more »

Transcription (biology)

Transcription is the first step of gene expression, in which a particular segment of DNA is copied into RNA (especially mRNA) by the enzyme RNA polymerase.

Autocrine signalling and Transcription (biology) · Benign prostatic hyperplasia and Transcription (biology) · See more »

The list above answers the following questions

Autocrine signalling and Benign prostatic hyperplasia Comparison

Autocrine signalling has 66 relations, while Benign prostatic hyperplasia has 130. As they have in common 3, the Jaccard index is 1.53% = 3 / (66 + 130).

References

This article shows the relationship between Autocrine signalling and Benign prostatic hyperplasia. To access each article from which the information was extracted, please visit:

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