Similarities between BRCA1 and MSH2
BRCA1 and MSH2 have 17 things in common (in Unionpedia): Ataxia telangiectasia and Rad3 related, BRCA1, Cancer, Caretaker gene, CHEK2, DNA repair, Epigenetics, Gene, MSH3, MSH6, Mutation, Nuclear localization sequence, P53, Protein, Protein dimer, Protein–protein interaction, Tumor suppressor.
Ataxia telangiectasia and Rad3 related
Serine/threonine-protein kinase ATR also known as ataxia telangiectasia and Rad3-related protein (ATR) or FRAP-related protein 1 (FRP1) is an enzyme that, in humans, is encoded by the ATR gene.
Ataxia telangiectasia and Rad3 related and BRCA1 · Ataxia telangiectasia and Rad3 related and MSH2 ·
BRCA1
BRCA1 and BRCA1 are a human gene and its protein product, respectively.
BRCA1 and BRCA1 · BRCA1 and MSH2 ·
Cancer
Cancer is a group of diseases involving abnormal cell growth with the potential to invade or spread to other parts of the body.
BRCA1 and Cancer · Cancer and MSH2 ·
Caretaker gene
Changes in the genome that allow uncontrolled cell proliferation or cell immortality are responsible for cancer.
BRCA1 and Caretaker gene · Caretaker gene and MSH2 ·
CHEK2
CHEK2 (Checkpoint kinase 2) is a tumor suppressor gene that encodes the protein CHK2, a serine-threonine kinase.
BRCA1 and CHEK2 · CHEK2 and MSH2 ·
DNA repair
DNA repair is a collection of processes by which a cell identifies and corrects damage to the DNA molecules that encode its genome.
BRCA1 and DNA repair · DNA repair and MSH2 ·
Epigenetics
Epigenetics is the study of heritable changes in gene function that do not involve changes in the DNA sequence.
BRCA1 and Epigenetics · Epigenetics and MSH2 ·
Gene
In biology, a gene is a sequence of DNA or RNA that codes for a molecule that has a function.
BRCA1 and Gene · Gene and MSH2 ·
MSH3
DNA mismatch repair protein, MutS Homolog 3 (MSH3) is a human homologue of the bacterial mismatch repair protein MutS that participates in the mismatch repair (MMR) system.
BRCA1 and MSH3 · MSH2 and MSH3 ·
MSH6
MSH6 or mutS homolog 6 is a gene that codes for DNA mismatch repair protein Msh6 in the budding yeast Saccharomyces cerevisiae.
BRCA1 and MSH6 · MSH2 and MSH6 ·
Mutation
In biology, a mutation is the permanent alteration of the nucleotide sequence of the genome of an organism, virus, or extrachromosomal DNA or other genetic elements.
BRCA1 and Mutation · MSH2 and Mutation ·
Nuclear localization sequence
A nuclear localization signal or sequence (NLS) is an amino acid sequence that 'tags' a protein for import into the cell nucleus by nuclear transport.
BRCA1 and Nuclear localization sequence · MSH2 and Nuclear localization sequence ·
P53
Tumor protein p53, also known as p53, cellular tumor antigen p53 (UniProt name), phosphoprotein p53, tumor suppressor p53, antigen NY-CO-13, or transformation-related protein 53 (TRP53), is any isoform of a protein encoded by homologous genes in various organisms, such as TP53 (humans) and Trp53 (mice).
BRCA1 and P53 · MSH2 and P53 ·
Protein
Proteins are large biomolecules, or macromolecules, consisting of one or more long chains of amino acid residues.
BRCA1 and Protein · MSH2 and Protein ·
Protein dimer
In biochemistry, a protein dimer is a macromolecular complex formed by two protein monomers, or single proteins, which are usually non-covalently bound.
BRCA1 and Protein dimer · MSH2 and Protein dimer ·
Protein–protein interaction
Protein–protein interactions (PPIs) are the physical contacts of high specificity established between two or more protein molecules as a result of biochemical events steered by electrostatic forces including the hydrophobic effect.
BRCA1 and Protein–protein interaction · MSH2 and Protein–protein interaction ·
Tumor suppressor
A tumor suppressor gene, or antioncogene, is a gene that protects a cell from one step on the path to cancer.
The list above answers the following questions
- What BRCA1 and MSH2 have in common
- What are the similarities between BRCA1 and MSH2
BRCA1 and MSH2 Comparison
BRCA1 has 157 relations, while MSH2 has 45. As they have in common 17, the Jaccard index is 8.42% = 17 / (157 + 45).
References
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