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Cannabinoid receptor antagonist and Mitogen-activated protein kinase

Shortcuts: Differences, Similarities, Jaccard Similarity Coefficient, References.

Difference between Cannabinoid receptor antagonist and Mitogen-activated protein kinase

Cannabinoid receptor antagonist vs. Mitogen-activated protein kinase

A cannabinoid receptor antagonist, also known simply as a cannabinoid antagonist or as an anticannabinoid, is a type of cannabinoidergic drug that binds to cannabinoid receptors (CBR) and prevents their activation by endocannabinoids. A mitogen-activated protein kinase (MAPK or MAP kinase) is a type of protein kinase that is specific to the amino acids serine and threonine (i.e., a serine/threonine-specific protein kinase).

Similarities between Cannabinoid receptor antagonist and Mitogen-activated protein kinase

Cannabinoid receptor antagonist and Mitogen-activated protein kinase have 1 thing in common (in Unionpedia): G protein–coupled receptor.

G protein–coupled receptor

G protein–coupled receptors (GPCRs), also known as seven-(pass)-transmembrane domain receptors, 7TM receptors, heptahelical receptors, serpentine receptor, and G protein–linked receptors (GPLR), constitute a large protein family of receptors that detect molecules outside the cell and activate internal signal transduction pathways and, ultimately, cellular responses.

Cannabinoid receptor antagonist and G protein–coupled receptor · G protein–coupled receptor and Mitogen-activated protein kinase · See more »

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Cannabinoid receptor antagonist and Mitogen-activated protein kinase Comparison

Cannabinoid receptor antagonist has 96 relations, while Mitogen-activated protein kinase has 136. As they have in common 1, the Jaccard index is 0.43% = 1 / (96 + 136).

References

This article shows the relationship between Cannabinoid receptor antagonist and Mitogen-activated protein kinase. To access each article from which the information was extracted, please visit:

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