Similarities between 5-HT2B receptor and Guanfacine
5-HT2B receptor and Guanfacine have 2 things in common (in Unionpedia): Alpha-2A adrenergic receptor, Valvular heart disease.
Alpha-2A adrenergic receptor
The alpha-2A adrenergic receptor (α2A adrenoceptor), also known as ADRA2A, is an α2 adrenergic receptor, and also denotes the human gene encoding it.
5-HT2B receptor and Alpha-2A adrenergic receptor · Alpha-2A adrenergic receptor and Guanfacine ·
Valvular heart disease
Valvular heart disease is any disease process involving one or more of the four valves of the heart (the aortic and bicuspid valves on the left side of heart and the pulmonary and tricuspid valves on the right side of heart. These conditions occur largely as a consequence of aging,Burden of valvular heart diseases: a population-based study. Nkomo VT, Gardin JM, Skelton TN, Gottdiener JS, Scott CG, Enriquez-Sarano. Lancet. 2006 Sep;368(9540):1005-11. but may also be the result of congenital (inborn) abnormalities or specific disease or physiologic processes including rheumatic heart disease and pregnancy. Anatomically, the valves are part of the dense connective tissue of the heart known as the cardiac skeleton and are responsible for the regulation of blood flow through the heart and great vessels. Valve failure or dysfunction can result in diminished heart functionality, though the particular consequences are dependent on the type and severity of valvular disease. Treatment of damaged valves may involve medication alone, but often involves surgical valve repair (valvuloplasty) or replacement (insertion of an artificial heart valve).
5-HT2B receptor and Valvular heart disease · Guanfacine and Valvular heart disease ·
The list above answers the following questions
- What 5-HT2B receptor and Guanfacine have in common
- What are the similarities between 5-HT2B receptor and Guanfacine
5-HT2B receptor and Guanfacine Comparison
5-HT2B receptor has 60 relations, while Guanfacine has 34. As they have in common 2, the Jaccard index is 2.13% = 2 / (60 + 34).
References
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