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Phagocyte and VDAC2

Shortcuts: Differences, Similarities, Jaccard Similarity Coefficient, References.

Difference between Phagocyte and VDAC2

Phagocyte vs. VDAC2

Phagocytes are cells that protect the body by ingesting harmful foreign particles, bacteria, and dead or dying cells. Voltage-dependent anion-selective channel protein 2 is a protein that in humans is encoded by the VDAC2 gene on chromosome 10.

Similarities between Phagocyte and VDAC2

Phagocyte and VDAC2 have 6 things in common (in Unionpedia): Apoptosis, Cell nucleus, Cytoplasm, Inflammation, Necrosis, Reactive oxygen species.

Apoptosis

Apoptosis (from Ancient Greek ἀπόπτωσις "falling off") is a process of programmed cell death that occurs in multicellular organisms.

Apoptosis and Phagocyte · Apoptosis and VDAC2 · See more »

Cell nucleus

In cell biology, the nucleus (pl. nuclei; from Latin nucleus or nuculeus, meaning kernel or seed) is a membrane-enclosed organelle found in eukaryotic cells.

Cell nucleus and Phagocyte · Cell nucleus and VDAC2 · See more »

Cytoplasm

In cell biology, the cytoplasm is the material within a living cell, excluding the cell nucleus.

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Inflammation

Inflammation (from inflammatio) is part of the complex biological response of body tissues to harmful stimuli, such as pathogens, damaged cells, or irritants, and is a protective response involving immune cells, blood vessels, and molecular mediators.

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Necrosis

Necrosis (from the Greek νέκρωσις "death, the stage of dying, the act of killing" from νεκρός "dead") is a form of cell injury which results in the premature death of cells in living tissue by autolysis.

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Reactive oxygen species

Reactive oxygen species (ROS) are chemically reactive chemical species containing oxygen.

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The list above answers the following questions

Phagocyte and VDAC2 Comparison

Phagocyte has 189 relations, while VDAC2 has 31. As they have in common 6, the Jaccard index is 2.73% = 6 / (189 + 31).

References

This article shows the relationship between Phagocyte and VDAC2. To access each article from which the information was extracted, please visit:

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